
Written by Robb Wolf (opens in a new tab)
Medically reviewed by Swapna Ghanta, MD (opens in a new tab) and Jared Meacham PhD., RD, CSCS (opens in a new tab)
Hypertension has been an established heart disease risk factorOpens in a new tab for decades. When blood flows at normal pressure, your vessels stay strong and flexible. Cranked up too high for a long period of time, this pressure can damage your vessel walls, making them stiff and narrow — ultimately increasing your risk for a cardiovascular event like heart attack or stroke.
Where does sodium come in? You likely learned in high school chemistry that this mineral, a component of salt, retains water. In your blood vessels, more volume means more pressure.
The U.S. government’s sodium intake recommendation of 2.3 grams per day leans heavily on this chemistry lesson, all in the name of heart health.
But this blanket recommendation is largely based on observational dataOpens in a new tab[*Opens in a new tab][*Opens in a new tab][*Opens in a new tab] — rather than controlled clinical studies — reporting higher sodium intakes are linked to higher blood pressure in certain populations.
The problem: Sodium also happens to be a critical component of heart health and overall well-being. Your body — and that includes your cardiovascular system — literally couldn’t function without it.
So it’s not surprising that other studies paint a different picture about this key electrolyte, linking higher salt intakes to less hypertensionOpens in a new tab and a lower risk of deathOpens in a new tab from stroke or heart attack.
Given the lack of large clinical trials forging a stronger link between sodium and heart disease risk, a 2020 studyOpens in a new tab goes as far as questioning blanket sodium intake recommendations, instead focusing on reducing processed foods that happen to be high in sodium. Of course, it’s always a good idea to check in with a trusted healthcare provider to understand and minimize your personal cardiovascular disease risk.
Sodium certainly isn’t a panacea — and more isn’t always better for every individual. But the latest nutritional science we’ll explore in this piece suggests that the USDA’s two gram recommendation is lower than what may be optimal, or even healthy for some.
Read on for a deep dive about sodium’s critical function in the body, and how you can leverage this electrolyte for your well-being.
Let’s get something straight: Sodium is not an optional mineral. Even low-salt advocates aren’t calling for zero sodium.
Every time the brain sends a signal through the nervous system, it does so using an action potential, or an electrical pulse. This pulse is made possible by sodium-potassium pumps that move sodium and potassium ions in and out of nerve cells to create the electrical charge needed for the signal.
The main functions of sodium in your body include:
At the bare minimum, everyone needs around 500 milligrams (mg) of sodium daily to squeak by (though the bare minimum says nothing about optimal intakes.)
If you don’t have the sodium you need to function — say your diet is very low in salt — your body actually goes into sodium-sparing mode. This is when certain hormones — such as aldosterone Opens in a new tab— kick up and you pee out less sodiumOpens in a new tab.
High-salt diets have the opposite effect, causing you to excrete more sodium, but we’ll get into that in a bit.
You might be wondering: If sodium is so important, what’s with the blanket recommendations against it?
It started with Lewis Dahl, a research scientist who published a number of papers in the 1960s and 1970s.
Back then, he discoveredOpens in a new tab that rats with certain genes — now called “Dahl rats” — would develop high blood pressure at high salt intakes. Dahl fed the rats the human equivalent of 560 grams of salt per day. That’s like ingesting a pound of salt in a 24-hour period. Not surprising, right? Chemistry 101.
Dahl also found a link between sodium and hypertension in human populations. The data was observational, however — and Dahl puzzledOpens in a new tab over the fact that many individuals on high-salt diets did not have high blood pressure.
Dahl’s findings were used, in part, to justify 1980 US Dietary Guidelines that admonishedOpens in a new tab the public to “avoid too much sodium.” These guidelines, however, didn’t say how much salt one should avoid.
This has changed. Today, the USDA recommends capping sodium at 2.3 grams per day to reduce the risk of hypertension and heart disease.
The American Heart Association (AHA) is even more bearish on salt, advising you to stay under 1.5 grams per day. For reference, the average American consumes about 3.4 grams of sodium per day — but it’s important to remember the average American also consumes a Standard America Diet (SAD) that’s heavy in processed, refined foods, which are often high in sodium.
A number of studiesOpens in a new tab (mostly observational, though some clinical) point to salt’s role in increasing blood pressure.
Physiologically, this isn’t surprising: If I inject you with loads of sodium, it will transiently increase your blood volume, and therefore your blood pressure.
But what about normal variations in dietary sodium intake? How does that affect blood pressure over time?
The data, to say the least, aren’t consistent.
Take the Intersalt StudyOpens in a new tab, published in 1988, which looked at sodium and blood pressure in over 10,000 people across 52 regions of the world.
In the vast majority of populations, researchers observed no link between sodium intake and high blood pressure. But some outlier populations stood out.
For instance, the Yanomami (native to Brazil) ate very little sodium and had — much to the glee of anti-salt folks — very low blood pressure.
To them, this correlation was data to vindicate low-salt diets. But the Yanomami aren’t exactly ideal models for the low-salt campaign.
In factOpens in a new tab, this population dies relatively young and appears to suffer from growth issues.
This isn’t to say inadequate sodium is the cause of their health woes, but it’s not exactly protecting them from the outcomes that matter most.
Since the Intersalt Study, evidence for sodium causing high blood pressure continues to underwhelm.
In 2017, researchers analyzedOpens in a new tab 2,632 people with normal blood pressure consuming either low (under 2.5 grams) or high (over 2.5 grams) sodium intakes. Guess what? The high sodium group had lower blood pressure than the salt restrictors. This simply couldn’t happen if more sodium always meant higher blood pressure.
Further, there may be some confounding factors affecting the conversation about salt and cardiovascular risk. As mentioned, SAD is ridden with processed foods that are also notoriously high in sodium.
Plus, people eating more sodium also tendOpens in a new tab to consume more sugary soft drinks.
Metabolic syndrome, which stems from insulin resistance — you guessed it, often as a result of sugary, processed foods — is another well-known risk factor for heart disease.
On the flipside, when people eat more whole foods, they naturally eat less sodium. They also tend to be people who exercise more — a lower-risk group when it comes to heart disease.
So could it be that salt is unfairly taking the heat, when metabolic syndrome is a far more pressing problem?
A subset of the population are genetically proneOpens in a new tab to hypertension at high salt intakes. We call these people “sodium hyper responders,” and their data is intermingled with everyone else’s — likely driving many positive correlations between sodium intake and blood pressure.
Should salt-sensitiveOpens in a new tab people avoid salt, then? Not necessarily. They may actually do better to increase potassiumOpens in a new tab — another crucial electrolyte abundant in fruits and vegetables.
When salt-sensitive people eat enough potassium (3.5–5 grams per day is a good evidence-basedOpens in a new tab target), it often neutralizesOpens in a new tab the effect of sodium on blood pressure. This effect also occurs, to a lesser extent, in the rest of the population.
Inadequate potassium could explain some data linking high-sodium diets to hypertension.
High-sodium diets tendOpens in a new tab to be full of refined foods, which are notoriously devoid of potassium.
Food quality matters tremendously, and it should be a far bigger part of the heart health conversation than it is.
The case for sodium restriction lowering blood pressure is tenuous at best. But even if we buy that argument for a moment, what about the outcomes that really matter? Do low-sodium dietsOpens in a new tab actually prevent heart attacks, strokes, and cardiac death?
In my opinion, the best answer to this question was publishedOpens in a new tab in the Journal of the American Medical Association (JAMA).*
Researchers followed 4,729 heart disease patients over several years, routinely measuring sodium excretion as a proxy for sodium intake.
When the study wrapped, 2,057 of the patients had died from stroke, heart attack, and other cardiac-related deaths. Researchers then plotted the risk of death data against the sodium intake data:

As you can see, the lowest risk of death (bottom of the U) aligns with about 4–6 grams of sodium excretion per day. Travel left from this spot to lower sodium excretion and the death risk shoots up. Note, however, that sodium intake and excretion are different, and we aren’t able to pinpoint the sodium intake of the study participants. So this study’s findings should be taken with a grain of, well, salt.
More recently, researchers reviewedOpens in a new tab a number of clinical trials on low-sodium diets for treating heart failure. They found no solid evidence to support low-sodium diets.
At least according to this data, low-sodium diets don’t directly improve heart health in this population — and that’s why one-size-fits-all recommendations are likely not useful.
*This study sits within a broader discourse about how sodium intake is best measured. The Kawasaki equation is commonly used in large studies because it’s far easier than tracking exactly how much salt people eat and how much they lose. But it’s not perfect — it doesn’t capture the full picture, especially when it comes to how the body might be holding onto or losing sodium in ways that don’t show up in urine samples. Very few studies look at both intake and excretion together, largely because they’re expensive and complex to run. So while this method is widely accepted and the findings are helpful, they should still be interpreted with some caution.
Headaches, brain fog, fatigue, and irritabilityOpens in a new tab can be commonly caused by an electrolyte imbalance and sodium deficiencyOpens in a new tab.
When you restrict salt, your kidneys go into sodium-sparing mode. In other words, you pee out less sodium.
Think of it like wartime rations. When sodium is scarce, you save some for later. Your body is smart like that — hard-wired to survive scarcity of all types, be it food or sodium.
Preserving sodium, however, comes with consequences. Namely, a flood of hormones, including:
But the biggest danger of low-sodium diets? They increaseOpens in a new tab the risk of hyponatremiaOpens in a new tab (low blood serum sodium) and the muscle cramps, fatigue, seizures, confusion, and brain damage that often come along with it.
By itself, a low-sodium diet probably won’t cause hyponatremia. The main causes are:
But getting enough sodium gives you a baseline of protection, especially against the exercise-associated formOpens in a new tab of hyponatremia.
A few populationsOpens in a new tab in particular that must work extra hard to get enough sodium and avoid this condition include:
To determine if you might need more sodium, think through your daily routines and how you feel.
If you fall into any of the above categories and your performance is lagging, you’re having a tough time sleeping, or you feel dizzy when you stand up, supplementing sodium with an electrolyte powder, or smart dietary sources like olives or pickles may be a good idea.
I personally fall into three of the above categories, and for years I struggled to maintain my energy, both on and off the jiu jitsu mat.
Finally, with the help of my former coaches, I realized I wasn’t getting enough sodium. When I started taking a daily electrolyte powderOpens in a new tab, I felt like I’d jumped on a mushroom in Super Mario. (Not a drug reference, folks!)
So, what’s the right amount of sodium for you?
The government says to stay under 2.3 grams.
The average American eats about 3.4 grams.
And the sweet spot for outcomes may be closer to 4–6 grams of sodium excretion per day.
I’m not saying my story applies to everyone.We’re all different, and that’s why blanket recommendations aren't helpful. But when you look at the data on sodium, it seems to me that many people could do well with more salt, not less.
Always consult your doctor if you have serious medical conditions prior to starting any supplementation.
For more on my sodium (and other electrolyte) protocols, check out a couple of our other articles: